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  1. LittleTom

    Test Suspension

    Testosterone Suspension is an injectable preparation of pure, un-modified and un-esterified Testosterone, almost always suspended in a water base within microcrystals (hence the name Testosterone Suspension). Within the bodybuilding and athletic world, Testosterone Suspension is regarded as the most potent and strongest form of injectable Testosterone available, and it is known for producing some of the most rapid mass, strength, and physique changes out of all injectable preparations of Testosterone. This is for several reasons. The first reason is because this is the purest form of Testosterone, unmodified and unesterified, meaning it is instantly active the minute it is injected into the body. Also, because there is no added ester bonded to the Testosterone molecule, there is more total Testosterone per mg of Testosterone Suspension, making it a far more potent product. Because ester weights must be factored into the total weight of the substance, esterified formats of Testosterone (or any hormone) such as Testosterone Propionate, Testosterone Enanthate, and Testosterone Cypionate do not all yield 100mg of Testosterone in 100mg of Testosterone Enanthate, for example. Once the body cleaves off the ester that is bound to the molecule, the weight of the ester is removed, and so for example, in 100mg of Testosterone Enanthate, there is in reality approximately 70mg of Testosterone. 100mg of Testosterone Suspension, however, yields exactly 100mg of Testosterone. Because esterified variants of Testosterone have longer half-lives and require their esters to be removed (before releasing pure Testosterone), optimal peak blood plasma levels are achieved often in weeks of use. This is not the case with Testosterone Suspension, where optimal peak blood plasma levels are achieved in a matter of hours instead. For example, it takes approximately 4 or 5 weeks (some users even report as late as 6 weeks) of use of the longer Testosterone esters, such as Testosterone Enanthate and Cypionate, before mass and strength gains are experienced. With Testosterone Suspension, gains are normally experienced within the first week of use, and by 4 weeks into a Testosterone Suspension cycle, the bulk of the gains will usually have been achieved (meanwhile by this same time period, the longer acting formats of Testosterone will only just have begun taking effect). Testosterone Suspension is perhaps the oldest anabolic steroid preparation, first isolated and synthesized in the early 1930s by German scientists. It is the very first preparation of Testosterone to ever be created for use, and pre-dates the slower acting esterified variants of Testosterone by a matter of several years. Testosterone Propionate was developed towards the mid-1930s, and Testosterone Enanthate was available in the 1950s, but Testosterone Suspension still remained popular during this period of time even though it was widely regarded as a much more crude form of Testosterone that was inconvenient and uncomfortable to inject due to the painful injections, and the very short half-life that necessitated very frequent injections. By comparison, Testosterone Propionate required injections every other day to every four days, and Testosterone Enanthate required injections to be administered once or twice per week. Testosterone Suspension requires atleast daily injections, if not multiple times per day. Testosterone Suspension is the longest lasting Testosterone product on the prescription market, both in the USA and abroad and can be found under many American trade and brand names, such as Sterotate by Ulmer, Andronaq by Central, Aquasuspension Testosterone by Pitman-Moore, Injectable Aqueous Testosterone by Arlongton-Funk, Virosterone by Endo, and Testosterone Aqueous by National Drug Company. Testosterone Suspension is perhaps so widely available and manufactured in such numerous quantities and brands that there are countless amounts of brand names as well as generics. Clinical and medical application of Testosterone Susp was, of course, identical to that of all other Testosterone products. This included treatment of the following conditions: treatment for low libido (lack of sex drive), male impotence, hypogonadism and andropause (insufficient Testosterone production in males), the treatment of delayed-onset puberty in adolescent males, and even in females for the treatment of breast cancer as well as a few other conditions. Towards the late 1980s and 1990, the FDA, as it had done with all other anabolic steroids, amended the list of approved treatments by shortening it. Testosterone Suspension was then determined as a drug to focus almost exclusively on the treatment of hypogonadism and andropause, but is still today also reserved as a last resort therapy for the treatment of female breast cancer (though this is very rare considering the high incidence of virilization that Testosterone causes in females). Testosterone Suspension was in fact utilized quite extensively up until 1998 on the American prescription market. In 1998, Testosterone Suspension was primarily manufactured by Steris Laboratories in the United States, making it one of the last companies to manufacture the drug for medical use. Because of a small issue in regards to the dispensing of Schedule III drugs at the time, the FDA was forced to cause Steris to suspend production of all Schedule III drugs (including Testosterone Suspension) due to discrepancies in their inventory reports. It was not until several years down the line that Steris had the opportunity to pick up the manufacture of Testosterone Suspension once again, but they decided against it. Because of this, pharmaceutical grade Testosterone Susp is today available on the US prescription market only via private compounding pharmacies, making the drug a special-order item that can be difficult to acquire. It is also still widely available as a veterinary medicine. Because of the wide variation and selection of Testosterone Suspension throughout the world, the doses and concentrations it is available in varies greatly, with some products containing 100mg/ml or 50mg/ml (very common). Testosterone Suspension can be found contained in either 1ml ampoules or 10ml multidose vials. [h=2]Chemical Characteristics of Testosterone Suspension[/h] Testosterone Suspension is an injectable Testosterone product that contains free, pure, unmodified Testosterone in microcrystalline format that is suspended in a water base. Because there is no ester bond on the Testosterone molecule in this case, its half-life is greatly reduced compared to other injectable formats of Testosterone, making its half-life a matter of 2 – 4 hours (with some studies stating as high as 24 – 39 hours[1]). Once again, as stated earlier, it is important to understand that because Testosterone Suspension does not contain an carboxylic acid esterified to it, an individual using it is receiving far more Testosterone per mg of injection than with any other form of Testosterone. 100mg of Testosterone Suspension yields 100mg of Testosterone. By comparison, 100mg ofTestosterone Enanthate yields only 70mg of Testosterone (after the Enanthate ester has been removed by enzymes in the body, leaving free Testosterone). [h=2]Properties of Testosterone Suspension[/h] Testosterone has always been known to promote large amounts of nitrogen retention in muscle tissue, with studies indicating significant increases in fat-free mass and muscle size as a result[2]. Testosterone is also well-known for its ability to significantly increase levels of IGF-1 (Insulin-like Growth Factor 1) in muscle tissue, which further contributes to significant increases in muscle size and strength[3]. A study on Testosterone’s action within muscle tissue indicated that this primarily occurs through its ability to activate satellite cells in muscle tissue, which is very important in the role of repairing damaged muscle fibers[4]. That same study also indicated that Testosterone exhibits the ability to inhibit adipogenesis (the storage of fat) as well as the ability to increase the size of motor neurons. Testosterone has been found to achieve its muscle growth and strength-promoting effects primarily through the interaction with the androgen receptor located in muscle cells as one of its primary mechanisms[5]. Androgens such as Testosterone also increase red blood cell count via a stimulation of the increase of Erythropoietin in the kidneys, resulting in better oxygen transport throughout the body, thus increasing the endurance capabilities of the athlete[6].
  2. More than likely that answer is going to be some sort of testosterone. Which is definitely the most important or most needed addition to your regimen. It's the male dominated hormone. I can't imagine a blast or cruise without testosterone. So other than testosterone, which is your favorite or go to AAS?
  3. Guest

    Steroid ring guilty plea

    http://www.pressconnects.com/story/news/public-safety/2016/11/30/steroid-bust-guilty-plea-leader-distribution-group/94680400/
  4. dr.ziegler420

    Big Ramy

  5. dr.ziegler420

    Levrone never quits!!

  6. LittleTom

    Trenbolone Acetate

    [h=1]Trenbolone Acetate[/h] by Bill Roberts – Among the synthetic injectable anabolic steroids – compounds other than testosterone, the natural product – trenbolone is perhaps the most remarkable. Of them, it is certainly the most remarkable for mass gain, strength gain, and contest preparation. [h=3]Trenbolone Side Effects[/h] To some extent, there is a price in side effects to be paid for this, but not nearly to the extent that many imagine. While a few find trenbolone problematic for reasons of increased aggressive tendency, night sweats, or reduced aerobic performance, most find these side effects slight and easily managed, or non-existent for them personally. [h=3]Avoid Trenbolone-Only Cycles[/h] Trenbolone, whether as the acetate (Finaplix, Finajet), enanthate, or cyclohexylmethylcarbonate (Parabolan), should not be used as the sole anabolic in a steroid cycle. Mass gains are greatly limited when this is done. Instead, trenbolone should be stacked with Dianabol, Anadrol, or testosterone as the most common and highly suitable choices. [h=3]Trenbolone and Suppressed Estradiol Production[/h] A second reason is that estradiol levels fall too low when trenbolone is used alone. This occurs because natural testosterone production is suppressed when using trenbolone, and this in turn suppresses natural estradiol production. And since trenbolone itself does not aromatize (convert to estrogen), then there is very little substrate available for aromatization, and estrogen production becomes abnormally low. This can cause joint, mood, and libido problems. Concurrent use of testosterone, Dianabol, or HCG with trenbolone can solve the above estrogen issue. Typically at least 100 mg/week of testosterone will be used for this purpose, or at least 10 or 15 mg/day of Dianabol. [h=3]Stacking Trenbolone[/h] Trenbolone exhibits interesting stacking behavior. Combination with either Dianabol or Anadrol gives a very strong synergistic effect. Even if the same total milligram amount of steroid is used per week, results are much better with such a combination than with any of these steroids used alone. For example, 50 mg/day of trenbolone acetate used with 50 mg/day Dianabol gives much better results in a steroid cycle than either 100 mg/day TA used alone, or that amount of Dianabol used alone. I wouldn’t recommend either of those single-drug usages for a steroid cycle, but the trenbolone/Dianabol stack has produced amazing physique changes in very many instances. In contrast, trenbolone’s stacking behavior with oxandrolone (Anavar), methenolone (Primobolan), or drostanolone (Masteron) is entirely different. If keeping the same total milligram amounts, these combinations do not outperform trenbolone used alone. So, while it’s acceptable to combine these drugs, the purpose should not be to achieve increased total effect. Such combinations may be useful however to reduce side effects, or to accommodate the materials that are on hand. [h=3]Typical Trenbolone Dosages[/h] Trenbolone acetate is usually used at doses of 35-150 mg/day, and more typically 50-100 mg/day. The 35 mg figure generally is appropriate only when having high personal sensitivity to trenbolone-specific side effects. When trenbolone usage is this low and an effective cycle is desired, another injectable anabolic steroid should be added. Masteron is a good choice for this purpose. Another, quite different choice is testosterone. As for the higher 150 mg/figure, this generally is used for the purpose of increased nervous system stimulation compared to 100 mg/day rather than for further mass or strength improvements, which are already maximized or very nearly maximized at 100 mg/day. Most users find 50-75 mg/day to be an ideal dosage range, giving excellent benefit as part of an anabolic steroid stack. These milligram amounts are unusually low for an injectable anabolic steroid. Part of the reason is that trenbolone is remarkably potent (effective per milligram.) Another reason is that because the acetate ester is unusually light, a very high percentage of the weight of the trenbolone acetate molecule is the active steroid. [h=3]Trenbolone Acetate versus Trenbolone Enanthate[/h] In contrast, trenbolone enanthate’s percentage of active steroid is nearly 20% lower. For this reason, weekly total dosing for trenbolone enanthate is a little higher than for the acetate. For the enanthate, weekly total dosing is typically 300-800 mg. Unlike trenbolone acetate which has a half-life of only about 1 day and therefore should be injected daily, trenbolone enanthate is typically injected 2-4 times per week. The half life is probably about 5 days. [h=3]Trenbolone and Cortisol[/h] While the mass gain results from trenbolone can instead be achieved with high milligram amounts of other injectable anabolic steroids, trenbolone differs from them in being stronger as a glucocorticoid (cortisol) antagonist and in decreasing blood corticosteroid levels. For this reason, it reduces subcutaneous water retention. And further, this anti-cortisol effect can make a significant difference in fat loss. It may also be a partial explanation for any increased tendency to aggression that is sometimes seen with trenbolone. [h=3]Trenbolone and Progestagenic Activity?[/h] With regard to activity at other receptors, many have written regarding theories of progestagenic activity of trenbolone. Where legitimate trenbolone is used, practical experience shows no detectable effect with regard to water retention or gynecomastia. Veterinary literature reports no detectable progestagenic effect. And if this were not enough, recent cell culture research shows progestagenic potency to be only about 1% that of progesterone itself, which would not be an important amount. For these reasons, I would make no decisions against trenbolone from fears of claimed progestagenic activity. It is a very well-proven compound in bodybuilding, including in contest-prep situations where an actually-progestagenic compound would be very unsuited. [h=3]“Tren Coughâ€[/h] The final unusual thing about trenbolone to be mentioned is “tren cough.†This is a very brief episode which may occur with only a small percentage of injections, perhaps not even 1%. It can be very unpleasant for a minute or two. The most likely explanation is from irritant effect of a small amount of the oil preparation entering the bloodstream, not by direct injection into a vein but by leakage into a small blood vessel that was cut as the needle entered. There is no evidence of lasting harm from this.
  7. LittleTom

    Equipoise (bold)

    Equipoise® is the popularly referenced brand name for the veterinary injectable steroid boldenone undecylenate. Specifically it is a derivative of testosterone, which exhibits strong anabolic and moderately androgenic properties. The undecylenate ester greatly extends the activity of the drug (the undecylenate ester is only one carbon atom longer than decanoate), so that clinically injections would need to be repeated every three or four weeks. In veterinary medicine Equipoise© is most commonly used on horses, exhibiting a pronounced effect on lean bodyweight, appetite and general disposition of the animal. This compound is also said to shows a marked ability for increasing red blood cell production, although there should be no confusion that this is an effect characteristic of newly all anabolic/androgenic steroids. The favorable properties of this drug are greatly appreciated by athletes, Equipoise® being a very popular injectable in recent years. It is considered by many to be a stronger, slightly more androgenic Deca-Durabolin®. It is generally cheaper, and could replace Deca in most cycles without greatly changing the end result. The side effects associated with Equipoise® are generally mild. The structure of boldenone does allow it to convert into estrogen, but it does not have an extremely high affinity to do so. To try and quantify this we can look toward aromatization studies, which suggest that its rate of estrogen conversion should be roughly half that of testosterone's. The tendency to develop a noticeable amount of water retention with this drug would therefore be slightly higher than that with Deca-DurabolinO (with an estimated 20°/a conversion), but much less than what would be expected with a stronger agent such as Testosterone. While one does still have a chance of encountering an estrogen related side effect as such when using this substance, it is not a common problem when taken at a moderate dosage level. Gynecomastia might theoretically become a concern, but is usually only heaved of with very sensitive individuals or (again) those venturing high in dosage. Should estrogenic effects become troublesome, the addition of Nolvadex® and/or Proviron® should of course make the cycle more tolerable. An antiaromatase such as Cytadren® or Arimidex® would be stronger options, however probably not indicated with a mild drug as such. Equipoise® can also produce distinct androgenic side effects. Incidences of oily skin, acne, increased aggression and hair loss are likewise all possible with this compound, although will typically be related to the use of higher doses. Women in fact find this drug quite comfortable, virilization symptoms usually unseen when taken at low doses. Boldenone does reduce to a more potent androgen (dihydroboldenone) via the 5alpha reductase enzyme (which produces DHT from testosterone), however its affinity for this interaction in the human body is low to nonexistent". We therefore cannot consider the reductase inhibitor Proscar® to be of much use with Equipoise, as it would be blocking what is at best an insignificant path of metabolism for the steroid. And although this drug is relatively mild, it may still have a depressive effect on endogenous testosterone levels. A combination of HCG and Clomid®/Nolvadex® may likewise be needed at the conclusion of each cycle to avoid a "crash", particularly when running long in duration. Although it stays active for a much longer time, Equipoise® is injected at least once per week by athletes. It is most commonly used at a dosage of 200-400mg (4-8 ml, 50mg version) per week for men, 50-75 mg per week for women. Should a 25mg version be the only product available, the injection volume can become quite uncomfortable. The dosage schedule can be further divided, perhaps injections given every other day to reduce discomfort. One should also take caution to rotate injection sites regularly, so as to avoid irritation or infection. Should too large an oil volume be injected into one site, an abscess may form that requires surgical draining. To avoid such a problem, athletes will usually limit each injection to 3ml and reuse each site no more than once per week, preferably every other week. With Equipoise® this may require using not only the gluteus, but also the outer thighs for an injection site. Of course all problems associated with 25mg and 50mg dosed products are eliminated with the newer 100 mg and 200mg/ml versions of this steroid, which clearly give the user much more dosage freedom and injection comfort. Not a rapid mass builder, instead Equipoise® will be looked at to provide a slow but steady gain of strength and quality muscle mass. The most positive effects of this drug are seen when it is used for longer cycles, usually lasting more than 8-10 weeks in duration. The muscle gained should not be the smooth bulk seen with androgens, but very defined and solid. Since water bloat is not contributing greatly to the diameter of the muscle, much of the size gained on a cycle of Equipoise® can be retained after the drug has been discontinued. It is interesting to note that structurally Equipoise® and the classic bulking drug Dianabol are almost identical. In the case of Equipoise® the compound uses a l7beta ester (undecylenate), while Dianabol is 17 alpha alkylated. Aside from this the molecules are the same. Of course they act quite differently in the body, which goes to show the 17-methylation effects more than just the oral efficacy of a steroid. As discussed earlier, Equipoise® is a very versatile compound. We can create a number of drug combinations with it depending on the desired result. For mass, one may want to stack it with Anadrol 50®(oxymetholone) or an injectable testosterone such as Sustanon 250. The result should be an incredible gain of muscle size and strength, without the same intensity of side effects if using the androgen (at a higher dose) alone. During a cutting phase, muscle hardness and density can be greatly improved when combining Equipoise® with a non-aromatizable steroid such as trenbolone acetate, Proviron® (mesterolone; 1-methyl DHT), Halotestin® (fluoxymesterone), or Winstrol® (stanozolol). For some however, even the low buildup of estrogen associated with this compound is enough to relegate its use to bulking cycles only. Equipoise® is not an ideal steroid for the drug tested athlete however. This drug has the tendency to produce detectable metabolites in the urine months after use, a worry most commonly associated with Deca-Durabolin®. This is of course due to the high oil solubility of long chain esterified injectable steroids, a property which enables the drug to remain deposited in fatty tissues for extended periods of time. While this will reliably slow the release of steroid into the blood stream, it also allows small residual amounts to remain present in the body far after the initial injection. The release of stubborn stores of hormone would no doubt also be enhanced around contest time, a period when the athlete drastically attempts to mobilize unwanted body fat. If enough were used in the off-season, the athlete may actually fail a drug screen for boldenone although many months may have past since the drug was last injected.
  8. LittleTom

    Dianabol (D-Bol)

    Dianabol is the old Ciba brand name for the oral steroid methandrostenolone. It is a derivative of testosterone, exhibiting strong anabolic and moderate androgenic properties. This compound was first made available in 1960, and it quickly became the most favored and widely used anabolic steroid in all forms of athletics. This is likely due to the fact that it is both easy to use and extremely effective. In the U.S. Dianabol production had meteoric history, exploding for quite some time, then quickly dropping out of sight. Many were nervous in the late 80's when the last of the U.S. generics were removed from pharmacy shelves, the medical community finding no legitimate use for the drug anymore. But the fact that Dianabol has been off the U.S. market for over 10 years now has not cut its popularity. It remains the most commonly used black market oral steroid in the U.S. As long as there are countries manufacturing this steroid, it will probably remain so. Similar to testosterone and Anadrol 50®, Dianabol is a potent steroid, but also one which brings about noticeable side effects. For starters methandrostenolone is quite estrogenic. Gynecomastia is likewise often a concern during treatment, and may present itself quite early into a cycle (particularly when higher doses are used). At the same time water retention can become a pronounced problem, causing a notable loss of muscle definition as both subcutaneous water and fat build. Sensitive individuals may therefore want to keep the estrogen under control with the addition of an antiestrogen such as Nolvadex® and/or Proviron®. The stronger drug Arimidex® (antiaromatase) would be a better choice, but can also be quite expensive in comparison to standard estrogen maintenance therapies. In addition, androgenic side effects are common with this substance, and may include bouts of oily skin, acne and body/facial hair growth. Aggression may also be increased with a potent steroid such as this, so it would be wise not to let your disposition change for the worse during a cycle. With Dianabol there is also the possibility of aggravating a male pattern baldness condition. Sensitive individuals may therefore wish to avoid this drug and opt for a milder anabolic such as Deca-Durabolin®. While Dianabol does convert to a more potent steroid via interaction with the 5-alpha reductase anzyme (the same enzyme responsible for converting testosterone to dihydrotestosterone), it has extremely little affinity to do so in the human body's. The androgenic metabolite 5alpha dihydromethandrostenolone is therefore produced only in trace amounts at best. The benefit received from Proscar®/Propecia® would therefore be insignificant, the drug serving no real purpose. Being moderately androgenic, Dianabol is really only a popular steroid with men. When used by women, strong virilization symptoms are of course a possible result. Some do however experiment with it, and find low doses (5mg) of this steroid extremely powerful for new muscle growth. Whenever administered, Dianabol will produce exceptional mass and strength gains. In effectiveness it is often compared to other strong steroids like testosterone and Anadrol 50®, and it is likewise a popular choice for bulking purposes. A daily dosage of 4-5 tablets (20-25mg) is enough to give almost anybody dramatic results. Some do venture much higher in dosage, but this practice usually leads to a more profound incidence of side effects. It additionally adds well with a number of other steroids. It is noted to mix particularly well with the mild anabolic Deca-Durabolin®. Together one can expect an exceptional muscle and strength gains, with side effects not much worse than one would expect from Dianabol alone. For all out mass, a long acting testosterone ester like enanthate can be used. With the similarly high estrogenic/androgenic properties of this androgen, side effects may be extreme with such a combination however. Gains would be great as well, which usually makes such an endeavor worthwhile to the user. As discussed earlier, ancillary drugs can be added to reduce the side effects associated with this kind of cycle. In order to withstand oral administration, this compound is c17 alpha alkylated. We know that this alteration protects the drug from being deactivation by the liver (allowing nearly all of the drug entry into the bloodstream), however it can also be toxic to this organ. Prolonged exposure to c17 alpha alkylated substances can result in actual damage, possibly even the development of certain kinds of cancer. To be safe one might want to visit the doctor a couple of times during each cycle to keep an eye on their liver enzyme values. Cycles should also be kept short, usually less than 8 weeks long to avoid doing any noticeable damage. Jaundice (bile duct obstruction) is usually the first visible sign of liver trouble, and should be looked out for. This condition produces an unusual yellowing of the skin, as the body has trouble processing bilirubin. In addition to the skin, the whites of the eyes may also yellow, a clear indicator of trouble. Should this occur the drug should be discontinued immediately and a doctor visited. This is usually a point where further, permanent damage can be avoided. "It is also interesting to note that methandrostenolone is structurally identical to boldenone, except that it contains the added c17 alpha alkyl group discussed above. This fact makes clear the impact of altering a steroid in such a way, as these two compounds appear to act very differently in the body. The main dissimilarity seems to lie in the tendency for estrogenic side effects, which seems to be much more pronounced with Dianabol. Equipoise® is known to be quite mild in this regard, and users therefore commonly take this drug without any need to addition an antiestrogen. Dianabol is much more estrogenic not because it is more easily aromatized, as in fact the 17 alpha methyl group and c1-2 double bond both slow the process of aromatization. The problem is that methandrostenolone converts to l7alpha methylestradiol, a more biologically active form of estrogen than regular estradiol. But Dianabol also appears to be much more potent in terms of muscle mass compared to boldenone, supporting the notion that estrogen does play an important role in anabolism. In fact boldenone and methandrostenolone differ so much in their potencies as anabolics that the two are rarely though of as related. As a result, the use of Dianabol is typically restricted to bulking phases of training while Equipoise® is considered an excellent cutting or lean-mass building steroid. The half-life of Dianabol is only about 3 to 5 hours, a relatively short time. This means that a single daily dosage schedule will produce a varying blood level, with ups and downs throughout the day. The user likewise has a choice, to either split up the tablets during the day or to take them all at one time. The usual recommendation has been to divide them and try to regulate the concentration in your blood. This however, will produce a lower peak blood level than if the tablets were taken all at once, so there may be a trade off with this option. The steroid researcher Bill Roberts also points out that a single-episode dosing schedule should have a less dramatic impact on the hypothalamic-pituitary-testicular axis, as there is a sufficient period each day where steroid hormone levels are not extremely exaggerated. I tend to doubt hormonal stability can be maintained during such a cycle however, but do notice that anecdotal evidence often still supports single daily doses to be better for overall results. Perhaps this is the better option. Since we know the blood concentration will peak about 1.5 to 3 hours after administration, we may further wonder the best time to take our tablets. It seems logical that taking the pills earlier in the day, preferably some time before training, would be optimal. This would allow a considerable number of daytime hours for an androgen rich metabolism to heighten the uptake of nutrients, especially the critical hours following training.
  9. LittleTom

    Anadrol

    Anadrol 50 is the U.S. brand name for oxymetholone, a very potent oral androgen. This compound was first made available in 1960, by the international drug firm Syntex. Since oxymetholone is quite reliable in its ability to increase red blood cell production (and effect admittedly characteristic of nearly all anabolic/androgenic steroids), it showed particular promise in treating cases of severe anemia. For this purpose it turned out to be well suited, and was popular for quite some time. But recent years have brought fourth a number of new treatments, most notably the non-steroidal hormone Epogen (erythropoietin). This item is shown to have a much more direct effect on the red blood cell count, without the side effects of a strong androgen. Financial disinterest finally prompted Syntex to halt production of the U.S. Anadrol 50 in 1993, which was around the same time they decided to drop this item in a number of foreign countries. Plenastril from Switzerland and Austria was dropped; following soon was Oxitosona from Spain. Many Athletes feared Anadrol 50C~ might be on the way out for good. But new HIV/AIDS studies have shown a new light on oxymetholone. These studies are finding (big surprise) exceptional anti-wasting properties to the compound and believe it can be used safely in many such cases. Interest has been peaked, and as of 1998 Anadrol 50Â[emoji768] is again being sold in the United States. This time we see the same Anadrol 50Â[emoji768] brand name, but the manufacturer is the drug firm Unimed. Syntex continues to market license this drug in a number of countries however (under a few different brand names). Anadrol 50Â[emoji767] is considered by many to be the most powerful steroid available, with results of this compound being extremely dramatic. A steroid novice experimenting with oxymetholone is likely to gain 20 to 30 pounds of massive bulk, and it can often be accomplished in less than 6 weeks, with only one or two tablets per day. This steroid produces a lot of trouble with water retention, so let there be little doubt that much of this gain is simply bloat. But for the user this is often little consequence, feeling bigger and stronger on Anadrol 50 than any steroid they are l ikely to cross. Although the smooth look that results from water retention is often not attractive, it can aid quite a bit to the level of size and strength gained. The muscle is fuller, will contract better and is provided a level of protection in the form of "lubrication" to the joints as some of this extra water is held into and around connective tissues. This will allow for more elasticity, and will hopefully decrease the chance for injury when lifting heavy. It should be noted however, that on the other hand the very rapid gain in mass might place too much stress on your connective tissues for this to compensate. The tearing of pectoral and biceps tissue is commonly associated with heavy lifting while massing up on heavy androgens. There is such a thing as gaining too fast. Pronounced estrogen trouble also puts the user at risk for developing gynecomastia. Individuals sensitive to the effects of estrogen, or looking to retain a more quality look, will therefore often add NolvadexÂ[emoji768] to each cycle. It is important to note however, that this drug does not directly convert to estrogen in the body. Oxymetholone is a derivative of dihydrotestosterone, which gives it a structure that cannot be aromatized. As such, many have speculated as to what makes this hormone so troublesome in terms of estrogenic side effects. Some have suggested that it has progestational activity, similar to nandrolone, and is not actually estrogenic at all. Since the obvious side effects of both estrogens and progestins are very similar, this explanation might be a plausible one. However we do find medical studies looking at this possibility. One such tested the progestational activity of various steroids including nandrolone, norethandrolone, methandrostenolone, testosterone and oxymetholone 3. It reported no significant progestational effect inherent in oxymetholone or methandrostenolone, slight activity with testosterone and strong progestational effect inherent in nandrolone and norethandrolone. With such findings it starts to seem much more likely that oxymetholone can intrinsically activate the estrogen receptor itself, similar to but more profoundly than the estrogenic androgen methAndriol. In speaking with chemist Patrick Arnold about my thoughts on this, I was afforded very believable support for my suspected explanation. According to Pat: I share your thoughts on this. Anadrol has an acidic hydrogen in the A-ring at a vicinity that is approximate to where the acidic phenolic hydrogen of estradiol is. I suspect it is a potent estrogen agonist' Clearly if this is the case we can only combat the estrogenic side effects of oxymetholone with estrogen receptor antagonists such as NolvadexÂ[emoji768]or ClomidÂ[emoji768], and not with an aromatase inhibitor. The strong anti-aromatase compounds such as Cytadren and ArimidexÂ[emoji768] would similarly prove to be totally useless with this steroid, as aromatase is uninvolved. Anadrol 50 is also a very potent androgen. This trait tends to produce many pronounced, unwanted androgenic side effects. Oil skin, acne and body/facial hair growth can be seen very quickly with this drug. Many individuals respond with severe acne, often requiring medication to keep it under control. Some of these individuals find that Accutaine works well, which is a strong prescription drug that acts on the sebaceous glands to reduce the release of oils. Those with a predisposition for male pattern baldness may want to stay away from Anadrol 50 completely, as this is certainly a possible side effect during therapy. And while some very adventurous female athletes do experiment with this compound, it is much too androgenic to recommend. Irreversible virilization symptoms can be the result and may occur very quickly, possibly before you have a chance to take action. It is interesting to note that Anadrol 50 does exhibit some tendency to convert to dihydrotestosterone, although this does not occur via the 5-alpha reductase enzyme (responsible for altering testosterone to form DHT) as it is already a dihydrotestosterone based steroid. Aside from the added c-17 alpha alkylation (discussed below), oxymetholone differs from DHT only by the addition of a 2-hydroxymethylene group. This grouping can be removed metabolically however, reducing oxymetholone to the potent androgen l7alpha-methyl dihydrotestosterone (mesterolone; methyldihydrotestosterone)~. There is little doubt that this biotransformation contributes at least at some level to the androgenic nature of this steroid, especially when we note that in its initial state Anadrol 50 has a notably low binding affinity for the androgen receptor. So although we have the option of using the reductase inhibitor finasteride Proscar to reduce the androgenic nature of testosterone, it offers us no benefit with Anadrol 50Â[emoji768] as this enzyme is not involved. The principle drawback to Anadrol 50 is that it is a 17aipha alkylated compound. Although this design gives it the ability to withstand oral administration, it can be very stressful to the liver. Anadrol 50 is particularly dubious because we require such a high milligram amount per dosage. The difference is great when comparing it to other oral steroids like Dianabol orWinstrolwhich have the same chemical alteration. Since they have a slightly higher affinity for the androgen receptor, they are effective in much smaller doses (seen in the 5mg and 2mg tablet strengths). Anadrol 50 has a lower affinity, which may be why we have a 50mg tablet dosage. For comparison, taking three tablets of Anadrol 50 (150mg) is roughly the equivalent of 30 Dianabol tablets or 75 WinstrolÂ[emoji768] tablets(!). When looking at the medical requirements, the recommended dosage for all ages has been 1 - 5 mg/kg of body weight. This would give a 2201b person a dosage as high as 10 Anadrol 50 tablets (500mg) per day. There should be little wonder why when liver cancer has been linked to steroid use, Anadrol 50~ is generally the culprit. Athletes actually never need such a high dosage and will take in the range of only 1-3 tablets per day. Many happily find that one tablet is all they need for exceptional results, and avoid higher amounts. Cautious users will also limit the intake of this compound to no longer than 4-6 weeks and have their liver enzymes checked regularly with a doctor. Kidney functions may also need to be looked after during longer use, as water retention/high blood pressure can take a toll on the body. Before starting a cycle, one should know to give Anadrol 50 the respect it is due. It is a very powerful drug, but not always a friendly one.
  10. LittleTom

    Winstrol (stanozolo)

    Winstrol® is a popular brand name for the anabolic steroid stanozolol. This compound is a derivative of dihydrotestosterone, although its activity is much milder than this androgen in nature. It is technically classified as an anabolic steroid, shown to exhibit a slightly greater tendency for muscle growth than androgenic activity in early studies. While dihydrotestosterone really only provides androgenic side effects when administered, stanozolol instead provides quality muscle growth. Admittedly the anabolic properties of this substance are still mild in comparison to many stronger compounds, but it is still a reliable builder. Its efficacy as an anabolic could even be comparable to Dianabol, however Winstrol® does not carry with it the same tendency for water retention. Stanozolol also contains the same c17 methylation we see with Dianabol, an alteration used so that oral administration is possible. To spite this design however, there are many injectable versions of this steroid produced. Structurally stanozolol is not capable of converting into estrogen. Likewise an antiestrogen is not necessary when using this steroid, gynecomastia not being a concern even among sensitive individuals. Since estrogen is also the culprit with water retention, instead of bulk Winstrol® produces a lean, quality look to the physique with no fear of excess subcutaneous fluid retention. This makes it a favorable steroid to use during cutting cycles, when water and fat retention are a major concern. It is also very popular among athletes in combination strength/speed sports such as Track and Field. In such disciplines one usually does not want to carry around excess water weight, and may therefore find the raw muscle-growth brought about by Winstrol® quite favorable over the lower quality mass gains of more estrogenic agents. Have been noticed when trying to administer these products, even when using a large 22-gauge needle. But there are both advantages and disadvantages to each type of product. On the one hand the large particle size would form a longer acting deposit (depot) while the steroid dissolves, giving us the option of fewer injections. A larger shot every three to four days would likely be sufficient to keep blood levels within limits, which is a favorable schedule for a water-based product. On the other hand we are forced to use a standard size oil needle (21-22 gauge) for the injection, uncomfortable for regular administration. Products made with a finer substance do not allow for as slow acting a depot and therefore are usually injected every other day to keep blood levels steady. But shots can be given with a much more comfortable sized needle, opening up many new injection sites. Although you can jam a big "oil pipe" into your shoulder, it is really not the place for it. For men the usual dosage of Winstrol® is 15-25mg per day for the tablets and 25-50mg per day with the injectable (differences based solely on price and quantity). It is often combined with other steroids depending on the desired result. For bulking purposes, a stronger androgen like testosterone, Dianabol or Anadrol 50® is usually added. Here Winstrol® will balance out the cycle a bit, giving us good anabolic effect with lower overall estrogenic activity than if taking such steroids alone. The result should be a considerable gain in new muscle mass, with a more comfortable level of water and fat retention. For contest and dieting phases we could alternately combine Winstrol® with a non-aromatizing androgen such as trenbolone or Halotestin®. Such combinations should help bring about the strongly defined, hard look of muscularity so sought after among bodybuilders. Older, more sensitive individuals can otherwise addition compounds like Primobolan®, Deca-Durabolin® or Equipoise® when wishing to stack this steroid. Here we should see good results and fewer side effects than is to be expected with standard androgen therapies. Women will take somewhere in the range of 5-l0mg daily, or two and a half to five 2mg tablets. Although female athletes usually find stanozolol very tolerable, the injectable is usually off limits. They risk androgenic buildup, as a regular 50mg injection will provide much too high a dosage. Here the tablets are the general preference. It is obviously much easier to divide up pills than it is to break up a 1cc ampule into multiple injections. Those who absolutely must experiment with the injectable would be most comfortable dividing each 50mg ampule into at least two separate injections. At this point the dosage will adjusted by the number of days separating each shot. 25mg every third or fourth day should be a comfortable amount for most. More ambitious (and risk taking) females would take 25mg every second day, although this is not recommended. Although this compound is only moderately androgenic, the risk of virilization symptoms should remain a concern. With the structural (c17-AA) alteration, the tablets will also place a higher level of stress on the liver than the injectable (which avoids the "first pass"). During longer or higher dosed cycles, liver values should therefore be watched closely through regular blood work. Although less common, the possibility of liver damage cannot be excluded with the injectable however. While it does not enter the body through the liver, it is still broken down by it, providing a lower (but more continuous) level of stress. Such stress would of course be amplified when adding other c17-AA oral compounds to a cycle of Winstrol®. When using such combinations, cautious users would make every effort to limit the length of the cycle (preferably 6 to 8 weeks). It is also of note that both versions of Winstrol® have been linked to strong adverse changes in HDULDL cholesterol levels. This side effect is common with anabolic steroid therapy, and obviously can become a health concern as the dose/duration of intake increase above normal. The oral version should have a greater impact on cholesterol values than the injectable due to the method of administration, and may therefore be the worse choice of the two for those concerned and this side effect. As discussed in the opening section of this book, the oral use of stanozolol can also have a profound impact on levels of SHBG (sex hormone-binding globulin). This admittedly is characteristic of all anabolic/androgenic steroids, however its potency and form of administration make Winstrol® particularly noteworthy in this regard. Since plasma binding proteins such as SHBG act to temporarily constrain steroid hormones from exerting activity, this effect would provide a greater percentage of free (unbound) steroid hormone in the body. This may amount to an effective mechanism in which stanozolol could increase the potency of a concurrently used steroid. To further this purpose we could also addition Proviron® (1 methyl-dihydrotestosterone), which has an extremely high affinity for SHBG. This affinity may causeProviron® to displace other weaker substrates for SHBG (such as testosterone), another mechanism in which the free hormone level may be increased. Adding Winstrol® andProviron® to your next testosterone cycle may therefore prove very useful,, markedly enhancing the free state of this potent muscle building androgen.
  11. LittleTom

    Anavar

    Anavar was the old U.S. brand name for the oral steroid oxandrolone, first produced in 1964 by the drug manufacturer Searle. It was designed as an extremely mild anabolic, one that could even be safely used as a growth stimulant in children. One immediately thinks of the standard worry, "steroids will stunt growth". But it is actually the excess estrogen produced by most steroids that is the culprit, just as it is the reason why women stop growing sooner and have a shorter average stature than men. Oxandrolone will not aromatize, and therefore the anabolic effect of the compound can actually promote linear growth. Women usually tolerate this drug well at low doses, and at one time it was prescribed for the treatment of osteoporosis. But the atmosphere surrounding steroids began to change rapidly in the 1980's, and prescriptions for oxandrolone began to drop. Lagging sales probably led Searle to discontinue manufacture in 1989, and it had vanished from U.S. pharmacies until recently. Oxandrolone tablets are again available inside the U.S. by BTG, bearing the new brand name Oxandrin. BTG purchased rights to the drug from Searle and it is now manufactured for the new purpose of treating HIV/AIDS related wasting syndrome. Many welcomed this announcement, as Anavar had gained a very favorable reputation among athletes over the years. Anavar is a mild anabolic with low androgenic activity. Its reduced androgenic activity has much to due 4vith the fact that it is a derivative of dihydrotestosterone. Although you might think at first glance this would make it a more androgenic steroid, it in fact creates a steroid that is less androgenic because it is already "5-alpha reduced". In other words, it lacks the capacity to interact with the 5-alpha reductase enzyme and convert to a more potent "dihydro° form. It is a simply matter of where a steroid is capable of being potentiated in the body, and with oxandrolone we do not have the same potential as testosterone, which is several times more active in androgen responsive tissues compared to muscle tissue due to its conversion to DHT. It essence oxandrolone has a balanced level of potency in both muscle and androgenic target tissues such as the scalp, skin and prostate. This is a similar situation as is noted with Primobolan and Winstrol, which are also derived from dihydrotestosterone yet not known to be very androgenic substances. This steroid is known as a good agent for the promotion of strength and duality muscle mass gains, although the mild nature of this compound makes it less than ideal for bulking purposes. Among bodybuilders it is most commonly used during cutting phases of training when water retention is a concern. The standard dosage for men is in the range of 15-25mg (6-10 tablets) per day, a level that should produce noticeable results. It can be further combined with anabolics like Primobolan® and Winstrol® to elicit a harder. more defined look without added water retention. Such combinations are very popular and can dramatically enhance the show physique. One can also add strong non-aromatizing androgens like Halotestin®, Proviron® or trenbolone. In this case the androgen really helps to harden up the muscles, while at the same time making conditions more favorable for fat reduction. Some athletes do choose to incorporate oxandrolone into bulking stacks. but usually with standard bulking drugs like testosterone or Dianabol. The usual goal in this instance is an additional gain of strength, as well as more quality look to the androgen bulk. Women who fear the masculinizing effects of many steroids would be quite comfortable using this drug, as this is very rarely seen with low doses. Here a daily dosage of 5mg should illicit considerable growth without the noticeable androgenic side effects of other drugs. Eager females may wish to addition mild anabolics like Winstrol®, Primobolan® or Durabolin®. When combined with such anabolics, the user should notice faster, more pronounced muscle-building effects, but may also increase the likelihood of androgenic buildup. Studies using low dosages of this compound note minimal interferences with natural testosterone production. Likewise when it is used alone in small amounts there is typically no need for ancillary drugs like Clomid®/Nolvadex® or NCG. This has a lot to do with the fact that it does not convert to estrogen, which we know has an extremely profound effect on endogenous hormone production. Without estrogen to trigger negative feedback, we seem to note a higher threshold before inhibition is noted. But at higher dosages of course, a suppression of natural testosterone levels will still occur with this drug as with any anabolic/androgenic steroid. This makes clear that while estrogen is important in this regard, androgen action triggers feedback inhibition as well. In the context of the average bodybuilder using this steroid at a level to promote growth, we would probably expect that maintaining a normal level of endogenous testosterone release would likewise be very difficult. Anavar is also a 17alpha alkylated oral steroid, carrying an alteration that is noted for putting stress on the liver. It is importar7t to point out however that to spite this alteration oxandrolone is generally very well tolerated, While liver enzyme tests will occasionally show elevated values, actual damage due to this steroid is not a statistical problem. Bio-Technology General states that oxandrolone is not as extensively metabolized by the liver as other l7aa orals are; evidenced by the fact that nearly a third of the compound is still intact when excreted in the urine. This may have to do with the understood milder nature of this agent (compared to other l7aa orals) in terms of hepatotoxicity. One study comparing the effects of oxandrolone to other agents including as methyltestosterone, norethandrolone, fluoxymesterone and methAndriol clearly supports this notion45. Here it was demonstrated that oxandrolone causes the lowest sulfobromophthalein (BSP; a marker of liver stress) retention among al! the alkylated orals tested. 20mg of oxandrolone in fact produced 72% less BSP retention than an equal dosage of fluoxyrnesterone, which is a considerable difference being that they possess the same liver-toxic alteration. With such findings, combined with the fact that athletes rarely report trouble with this drug, most feel comfortable believing it to be much safer to use during loner cycles than most of other orals with this distinction. Although this may very well be true, the chance of liver damage still cannot be excluded however. At one time oxandrolone was also looked at as a possible drug for those suffering from disorders of high cholesterol or trigfycerides. Ear(y studies showed it to be capable of lowering total cholesterol and triglyceride values in certain types of hyperlipidemic patients, which initially this was thought to signify potential for this drug as a hypo-lipid (lipid lowering) agent'°. With further investigation we find however that while use of this drug can be linked to a lowering of total cholesterol values, it is such that a redistribution in the ratio of good (HDL) to bad (LDL) cholesterol occurs, usually moving values in an unfavorable direction4' 48. This would of course negate any positive effect that the drug might have on triglycerides or total cholesterol, and in fact make it a danger in terms of cardiac risk when taken for prolonged periods of time. Today we understand that as a group anabolic/androgenic steroids produce very unfavorable changes in lipid profiles, and are really not useful in disorders of lipid metabolism. As an oral c17 alpha alkylated steroid, oxandrolone is probably even more risky to use than an injectable esterified injectable such as a testosterone or nandrolone in this regard.
  12. LittleTom

    Deca Durabolin

    Deca-Durabolin® is the Organon brand name for the injectable steroid nandrolone decanoate. This compound came around early in the wave of commercial steroid development, first being made available as a prescription medication in 1962. This steroid is an extremely long acting compound, with the decanoate ester said to provide this drug a slow release time of up to three or four weeks. While perhaps true in a technical sense, what we find with further investigation is that the release parameters after a single injection are such that a strong release of nandrolone is really only maintained for one to two weeks. This figure admittedly fails to take into account drug buildup that may occur after multiple injections, which may allow a longer duration of good effect to be seen. Figure 1 is provided to illustrate the release dynamics of a single 200mg injection. As you will see, by the end of the second week levels are already approaching baseline. World Wide "Deca" is one of the most widely used anabolic steroids. Its popularity is due to the simple fact that it exhibits many very favorable properties. Structurally nandrolone is very similar to testosterone, although it lacks a carbon atom at the 19t" position (hence its other name 19-nortestosterone). The resulting structure is a steroid that exhibits much weaker androgenic properties than testosterone. Of primary interest is the fact that nandrolone will not break down to a more potent metabolite in androgen target tissuess'. You may remember this is a significant problem with testosterone. Although nandrolone does undergo reduction via the same (5-alpha reductase) enzyme that produces DHT from testosterone, the result in this case is dihydronandrolone. This metabolite is weaker than the parent nandroloness, and is far less likely to cause unwanted androgenic side effects. Strong occurrences of oily skin, acne, body/facial hair growth and hair loss occur very rarely. It is however possible for androgenic activity to become apparent with this as any steroid, but with nandrolone higher than normal doses are usually responsible. Nandrolone also show an extremely lower tendency for estrogen conversion. For comparison, the rate has been estimated to be only about 20% of that seen with testosterones9. This is because while the liver can convert nandrolone to estradiol, in other more active sites of steroid aromatization such as adipose tissue nandrolone is far less open to this process'°. Consequently estrogen related side effects are a much lower concern with this drug. An antiestrogen is likewise rarely needed with Deca, gynecomastia only a worry among sensitive individuals. At the same time water retention is not a usual concern. This effect can occur however, but is most often related to higher dosages. The addition of Proviron® and/or Nolvadex® should prove sufficient enough to significantly reduce any occurrence. Clearly Deca is a very safe choice among steroids. Actually, many consider it to be the best overall steroid for a man to use when weighing the side effects and results. It should also be noted that in HIV studies, Deca has been shown not only to be effective at safely bringing up the lean body weight of patient, but also to be beneficial to the immune system. It is of note however that nandrolone is believed to have some activity as a progestin in the body". Although progesterone is a c-19 steroid, removal of this group as in 19-norprogesterone creates a hormone with greater binding affinity for its corresponding receptor. Sharing this trait, many 19-nor anabolic steroids are shown to have some affinity for the progesterone receptor as well'2. This can lead to some progestin-like activity in the body, and may intensify related side effects. The side effects associated with progesterone are actually quite similar to those of estrogen, including negative feedback inhibition of testosterone production, enhanced rate of fat storage and possibly gynecomastia. Many believe the progestin activity of Deca notably contributes to suppression of testosterone synthesis, which can be marked despite a low tendency for estrogen conversion. Deca is not known as a very "fast" builder. The muscle building effect of this drug is quite noticeable, but not dramatic. The slow onset and mild properties of this steroid therefore make it more suited for cycles with a longer duration. In general one can expect to gain muscle weight at about half the rate of that with an equal amount of testosterone. A cycle lasting eight to twelve weeks seems to make the most sense, expecting to elicit a slow, even gain of quality mass. Although active in the body for much longer, Deca is usually injected once per week. The dosage for men is usually in the range of 200-600mg. If looking to be specific, it is believed that Deca will exhibit its optimal effect (best gain/side effect ratio) at around 2mg per pound of bodyweight/weekly. Deca is also a popular steroid among female bodybuilders. They take a much lower dosage on average than men of course, usually around 50mg weekly. Although only slightly androgenic, women are occasionally confronted with virilization symptoms when taking this compound. Should this become a concern, the shorter acting nandrolone Durabolin® would be a safer option. This drug stays active for only a few days, greatly reducing the impact of androgenic buildup if withdrawal were indicated. "As mentioned earlier, endogenous testosterone levels can be a concern with Deca-Durabolin®, especially after long cycles. It is therefore a good idea to incorporate ancillary drugs at the conclusion of therapy. An estrogen antagonist such as Clomid® or Nolvadex® is therefore commonly used for a few weeks. These both provide a good level of testosterone stimulation, although they may take a couple of weeks to show the best effect. HCG injections could be added for extra reassurance, acting to rapidly restore the normal ability of the testes to respond to the resumed release of gonadotropins. For this purpose one could administer three injections of 2500-50001.U., spaced five days apart. After which point the antiestrogen is continued alone for a few more weeks in an effort to stabilize the production of testosterone. Remember to begin the ancillaries after Deca has been withdrawn for a few weeks, not the first week after the last shot. Deca stays active for quite some time so the ancillary drugs will not be able to exhibit their optimal effect when the steroid is still being released into the bloodstream. The major drawback for competitive purposes is that in many cases nandrolone metabolites will be detectable in a drug screen for up to a year (or more) after use. This is clearly due to the form of administration. As discussed earlier in this book, esterified compounds have a high affinity to stay stored in fatty tissues. While we can accurately estimate the time frame it will take for a given dose to enter circulation from an injection site, we cannot know for sure that 100% of the steroid will have been metabolized at any given point. Small amounts may indeed be stubborn in leaving fatty tissue, particularly after heavy, longer-term use. Some quantity of nandrolone decanoate may therefore be left to sporadically enter into the blood stream many months after use. This process may be further aggravated when dieting for a show, a time when body fat sores are being actively depleted (possibly freeing more steroid). This has no doubt been the cause for many unexpected positives on a drug screen. The fact that nandrolone has been isolated as the "hands-off" injectable for the drug tested athlete is most likely due to its popularity (and therefore common appearance on drug screens). The same risk would of course hold true for other long chain esterified injectables such as Equipoise®,Parabolan® and Primobolan®.On the other hand we find that the use of the oral nandrolone precursors norandrostenedione and norandrostenediol can allow the drug-tested athlete the benefit of an injectable nandrolone, without the same risk for a positive result. A recently published French study makes this possibility very clear. During this investigation it was shown that trace levels of the nandrolone metabolites norandrosterone and noretiocholanolone could be found in human urine up to eight months after a single 50mg injection of nandrolone undecanoate". This time frame shrank to only 8 days with norandrostenediol (50mg) and norandrostenedione (100mg). I have also had the opportunity to speak with an amateur bodybuilder recently, who was unexpectedly subject to a drug screen and now strongly supports the use of oral precursor hormones. He was using up to 3 grams norandrostenedione daily not very far from the date of the show, and to his amazement did not test positive for steroid use.
  13. Guest

    Trestolone( ment)

    Trestolone ace, more popularly known as Ment, has developed quite a reputation over the last several years for being an exceedingly potent steroid. Combined with its initial long-term absence from the market, its current limited availability, and a market nearly devoid of high quality clones, its allure among steroid users has been further increased, taking on an almost super-drug status similar to what we saw when the now defunct Parabolan ceased production. However, unlike most of the AAS which have hit the market in the last several years, Ment does not owe its popularity to enterprising OTC supplement companies or blackmarket dealers, but to legitimate medical research which has made its way into the BB’ing community. Ment was originally developed in the 1960’s, but recent interest has been generated by both the Population Council and the research-based pharmaceutical company Schering, due to their extensive investigation into Ment for the purposes of male birth control and hormone replacement therapy. This has resulted in some impressive findings, leading to the discovery of a steroid displaying a unique embodiment of traits, which are ideally suited to the muscle building process. Belonging to the 19-nor family of steroids, Ment is more properly known as 7-alpha-methyl-nortestosterone, yet despite its close association to this class of steroids, Ment displays several characteristics more commonly attributed to Testosterone. Nowhere is this more evident than in the area of male sexual functioning. Anyone who has been around the steroid subculture long enough is aware that the 19-nor family of drugs is notorious for causing sexual dysfunction in males. Side effects such as low libido and/or the inability to obtain an erection characterize the use of steroids like Nandrolone or Trenbolone and led to the coining of terms such as “Deca-dick†and “Tren-dickâ€. While the humor inherent in such language is apparent, those who suffer with these self-inflicted maladies generally don’t find it a laughing matter. This is where Ment differs. In fact, Ment is the only steroid in production today that is capable of sustaining normal male physiology in the complete absence of testosterone, including sexual functioning. This is one of the vital traits which has qualified Ment for consideration as a male contraceptive and hormone replacement therapy, as sexual dysfunction would be an unacceptable side effect in users seeking medical treatment for these reasons. With regard to cycle set-up, the inclusion of Ment allows for the revision of one of its most basic tenants, which traditionally says that testosterone should be included in every cycle. Although I personally do not agree with that philosophy in all cases, there are many good reasons why Testosterone should generally be included in one’s cycle. However, Ment changes all that, making the inclusion of Testosterone optional and not a necessity. All the side effects which normally present themselves in a state of Testosterone deficiency are absent when Ment is employed. This is a big advantage unique to Ment alone and which enables an AASA user to think outside the box when designing his/her cycle. Ment demonstrates a strong binding affinity for the AR receptor, being greater than that of Testosterone and even Nandrolone. Like Testosterone, it also has the ability to aromatize, making the concomitant use of an AI desirable in times of estrogen excess. As most AAS users are aware and which recent research confirms, estrogen plays a role in the muscle growth process both directly and indirectly, which is part of the reason why aromatizable steroids often impart superior mass gains compared to their non-aromatizing counterparts. So, while aromatization is beneficial in part, it can quickly be turned into a negative quality if not properly managed, resulting in side effects such as: gynecomastia, water retention, heightened blood pressure, increased HPTA suppression, mood swings, and the accumulation of additional bodyfat. Other defining characteristics of Ment include a lack of DHT conversion and like most injectable preparations, Ment is also non-hepatotoxic in nature. In addition, Ment does not bind to sex hormone binding globulin (SHBG), increasing the potency of this drug considerably. When administering drugs such as testosterone, over 95% of the injected steroid ultimately ends up either attaching to SHBG, converting to DHT, or aromatizing into estrogen, leaving only a small amount of the original dose left for muscle building functions. Once a hormone attaches to the protein SHBG, it remains bound (in most cases) for the entire life of the steroid, rendering it completely useless. AAS which avoid SHBG binding allow a significantly greater percentage of the injected drug to reach its intended target at the androgen receptor and initiate the muscle growth process. When determining Ment’s suitability as a prescription medication for male contraception or HRT, its effect on the prostate is of critical importance. This make or break factor was evaluated right from the start, with promising results. Research shows that Ment has a lesser effect on the prostate compared to testosterone per effective dose, reducing the potential risk of prostate issues in older men receiving treatment for HRT or otherwise. While younger users typically pay little heed to this aspect of their health, they may be glad they did later on down the road. One area where Ment isn’t quite so friendly is that of HPTA suppression. Research shows that Ment is a full 12X as suppressive as Testosterone on a mg to mg basis. However, this side effect is deemed to be a necessary benefit when looking to develop this drug as a birth control medication. Any steroid used for birth control purposes requires an exceptionally high success rate at preventing pregnancy, and that will only come by way of significant suppression of spermatogenesis. In reality, most steroid users end up largely infertile by cycle’s end anyway, due to the number and quantity of AAS used. Now let’s move onto the more exciting stuff; namely its ability to build muscle tissue. Most AAS users are primarily interested in one thing, which is…â€How much muscle will this stuff really help me add?†While this question is impossible to answer, due to the numerous influential variables involved, we can look to both medical research and real-world experience to help provide us with a clearer picture of what to expect. The following quote is an excerpt taken from a study comparing the effects of Testosterone and Trestolone: Ment delivers 10X the myotropic effect (muscle building) of testosterone, on a mg to mg basis. Based on these figures, this would make Ment more potent than any other non-toxic injectable currently sold on the market. I comparison to the exceedingly potent steroid Trenbolone, which is often used as a benchmark for potency, Ment outperforms it by a full 250%. It is important to note that the results witnessed in this study were obtained by using primates as test subjects, so while it is likely that the results will translate pretty well to humans, there is no substitute for real-world human testing being conducted specifically for the purpose of performance enhancement. The overwhelming majority of our BB’ing knowledge, as it relates to the optimal application of PED for the purpose of muscle growths, was gained through the combined experience of generations of athletes. While quality versions of this steroid are scarce, enough individuals have experimented with this steroid at this point in time, for us to have a good idea of what to expect. First of all, the BB’r should not expect to grow muscle at 10X the rate of Testosterone at an equivalent dose, but there is no doubt that one’s gains will measurably surpass what is attainable with an equal dose of Testosterone. Out of the few dozen people I have personally known who have used this steroid and based on the user experiences of others I am aware of, I would most aptly describe this steroid as a type of “super-testosteroneâ€, in terms of results. Visually, the musculature tends to take on a similar appearance. This is not a great steroid for contest prep or achieving a hard & dry look, but if sheer bulk accompanied by some water retention is what you’re looking for, Ment will deliver. Like Testosterone, estrogen-induced water retention can be substantially reduced with the concurrent use of an AI, allowing a decently hard & dry appearance to manifest. Even though Ment may behave similarly to Testosterone in terms of visual results & side effects, it is important to note that Ment’s ability to trigger these side effects at equivalent doses is much more exaggerated compared to testosterone. This is due largely to its increased androgenic potency. According to use feedback, Ment also carries with it the risk of developing certain “tren-like†sides in some users, especially at higher dosages, although not all users have reported this. As far as dosing is concerned, no definitive guidelines have yet been formed, although in my opinion it is likely that Ment will probably end up being dosed along the same lines as Trenbolone, with the average dose falling somewhere 200-500 mg per week. Of course, not all users will adhere to these guidelines, with some electing to administer a more hearty dosage. Overall, we are looking at a very potent steroid, both on paper and in the real-world, which is capable of eliciting serious gains in mass & strength over a relatively short period of time. As availability increases, look for this steroid to take up a more permanent residence in the arsenals of BB’rs and strength athletes alike.
  14. Finding the best and genuine can be tricky, I'll make it easier to you guys. Here are different sorts of body building supplements. Injectables Kratum Orals Peptides Bulk Powder Cycles & Stacks Sample Packs I hope these sources will help :)
  15. Guest

    FDA Steps up warning

    http://www.nbcnews.com/health/health-news/fda-steps-warnings-testosterone-other-steroids-n672681 The Food and Drug Administration said Tuesday it's stepping up warnings about testosterone and other steroid drugs. Not only can the drugs cause heart attacks, personality changes and infertility, but people can easily abuse them, the FDA said.
  16. Bama11

    History of steroids

    Whats up guys and gals at AA? I have been reading about the history of steroids and thought i would share a paragraph with you. Didnt know if you would be interested or not, if you have something to add or share please do. History The history of anabolic steroids can be traced back to as early as 1930's, before the term steroid was even used. In the 1930's, a team of scientists was able to create a synthetic form of testosterone (a male hormone) to help treat men who were unable to produce enough of the hormone for normal growth, development, and sexual functioning. Later, during World War II it was found that this artificial form of testosterone could be used to help malnourished soldiers gain weight and improve performance. After the war, athletes began to use steroids to enhance their performance in competitions. In the 1956 Olympics, Soviet athletes, especially wrestlers, performed at exceptionally high levels. After learning that those athletes were using testosterone, an American physician (Dr. Zeigler) created a more selective form of what we know as anabolic steroids. From that point until the early 1970's, steroids became increasingly popular among not just Olympic athletes, but also professional sports players and high school athletes. In 1975, the International Olympic Committee finally banned the use of steroids in Olympic competition. Black market (or illegal) sales continued to increase in the following years, and in 1988, the first major federal regulation of steroids was introduced as part of the Anti-Drug Abuse Act - stiffening penalties for the sale and possession of steroids. Only a couple of years later, Congress passed the Anabolic Steroid Enforcement Act of 1990, which placed certain anabolic steroids on Schedule III of the Controlled Substances Act (CSA). Previously, steroids had been unscheduled and controlled only by state laws. Today, illicit sales of steroids are still prevalent and surveys show that adolescent use of steroids is on the rise and that a great number of adults are actively using. Here is the artical where i found it enjoy!!! http://www.cesar.umd.edu/cesar/drugs/steroids.asp
  17. G_nome76

    BLAST

    Hey guys I'm currently on Test P 30-40mg eod and Tren A 150mg eod Thinking of a heavy blast as the kids are returning to there moms for the year and I will have time between now and the end of sept to train super hard. Here is what I'm thinking would love any and all feedback I do a lot of research but nothing beats battle tested knowledge. Test P 100mg ed Tren A 100 mg ed NPP 50mg ed Mast 75mg ed Eq 75mg ed Primo if I can get it 50-100mg ed Aromasin 12.5 Pramiplexole 2.5 Anavar 20 3xday Tren base for preworkout Am I missing anything or is any of it cancel out the others. From what I saw in research they should all work well together Sent from my iPhone using Tapatalk
  18. LittleTom

    Proviron

    Proviron® is the Schering brand name for the oral androgen mesterolone (1 methyl-dihydrotestosterone). Just as with DHT, the activity of this steroid is that of a strong androgen which does not aromatize into estrogen. In clinical situations Proviron® is generally used to treat various types of sexual dysfunction, which often result from a low endogenous testosterone level. It can usually reverse problems of sexual disinterest and impotency, and is sometimes used to increase the sperm count. The drug does not stimulate the body to produce testosterone, but is simply an oral androgen substitute that is used to compensate for a lack of the natural male androgen. Although this steroid is strongly androgenic, the anabolic effect of it is considered too weak for muscle building purposes. This is due to the fact that Proviron® is rapidly reduced to inactive metabolites in muscle tissue, a trait also characteristic of dihydrotestosterone, The belief that the weak anabolic nature of this compound indicated a tendency to block the androgen receptor in muscle tissue, thereby reducing the gains of other more potent muscle building steroids, should likewise not be taken seriously. In fact due to its extremely high affinity for plasma binding proteins such as SHBG, Proviron® may actually work to potentate the activity of other steroids by displacing a higher percentage into a free, unbound state. Among athletes Proviron® is primarily used as an antiestrogen. It is believed to act as an antiaromatase in the body, preventing or slowing the conversion of steroids into estrogen. The result is somewhat comparable to Arimidex® (though less profound), the drug acting to prevent the buildup of estrogen in the body. This is in contrast to Nolvadex®, which only blocks the ability of estrogen to bind and activate receptors in certain tissues. The anti-aromatization effect is preferred, as it is a more direct and efficient means of dealing with the problem of estrogenic side effects. A related disadvantage to Nolvadex® is that if discontinued too early, a rebound effect may occur as high serum estrogen levels are again free to take action. This of course could mean a rapid onset of side effects such as gynecomastia and water retention. Most athletes actually prefer to use both Proviron® and Nolvadex®, especially during strongly estrogenic cycles. With each item attacking estrogen at a different angle, side effects are often greatly minimized. The anti-estrogenic properties of Proviron® are not unique to this compound. A number of steroids have in fact demonstrated similar activity. Dihydrotestosterone and Masteron (2methyl-dihydrotestosterone) for example have been successfully used as therapies for gynecomastia and breast cancer due to their strong anti-estrogenic effect. It has been suggested that nandrolone may even lower aromatase activity in peripheral tissues where it is more resistant to estrogen conversion (the most active site of nandrolone aromatization seems to be the liver). The antiestrogenic effect of all of these compounds is presumably caused by their ability to compete with other substrates for binding to the aromatase enzyme. With the aromatase enzyme bound to the steroid, yet being unable to alter it, and inhibiting effect is achieved as it is temporarily blocked from interacting with other hormones. This drug is also favored by many during contest preparations, when a lower estrogen/high androgen level is particularly sought after. This is especially beneficial when anabolics likeWinstrol®, oxandrolone and Primobolan® are being used alone, as the androgenic content of these drugs is relatively low. Proviron® can supplement a wellneeded androgen, and bring about an increase in the hardness and density of the muscles. Women in particular find a single 25mg tablet will efficiently shift the androgen/estrogen ratio, and can have a great impact on the physique. Since this is such a strong androgen however, extreme caution should be taken with administration. Higher dosages clearly have the potential to cause virilization symptoms quite readily. For this reason females will rarely take more than one tablet per day, and limit the length of intake to no longer than four or five weeks. One tablet used in conjunction with 10 or 20mg of Nolvadex® can be even more efficient for muscle hardening, creating an environment where the body is much more inclined to burn off extra body fat (especially in female trouble areas like the hips and thighs). The typical dosage for men is one to four 25 mg per tablets per day. This is a sufficient amount to prevent gynecomastia, the drug often used throughout the duration of a strong cycle. As mentioned earlier, it is often combined with Nolvadex® (tamoxifen citrate) or Clomid® (clomiphene citrate) when heavily estrogenic steroids are being taken (Dianabol, testosterone etc.). Administering 50mg of Proviron® and 20mg Nolvadex® daily has proven extremely effective in such instances, and it is quite uncommon for higher dosages to be required. And just as we discussed for women, the androgenic nature of this compound is greatly welcome during contest preparation. Here again Proviron® should noticeably benefit the hardness and density of the muscle, while at the same time increasing the tendency to burn off a greater amount of body fat. Proviron® is usually well tolerated and side effects (men) are rare with dosages under 100 mg per day. Above this, one may develop an excessively high androgen level and encounter some problems. Typical androgenic side effects include oily skin, acne, body/facial hair growth and exacerbation of a male pattern baldness condition, and may occur even with the use of a moderate dosage. With the strong effect DHT has on the reproductive system, androgenic actions may also include an extreme heightening of male libido. And as discussed earlier, Women should be careful around Proviron®. It is an androgen, and as such has the potential to produce virilization symptoms quite readily. This includes, of course, a deepening of the voice, menstrual irregularities, changes in skin texture and clitoral enlargement. Proviron® is also not a c17 alpha alkylated compound, an alteration commonly used with oral anabolic/androgenic steroids. Not using this structure in the case of Proviron® removes the notable risk of liver toxicity we normally associate with oral dosing. We therefore consider this a "safe" oral, the user having no need to worry about serious complications with use. This steroid in fact utilizes the same 1-methylation we see present on Primobolan® (methenolone), another well tolerated orally active compound. Alkylation at the one position also slows metabolism of the steroid during the first pass, although much less profoundly than 17 alpha alkylation. Likewise Proviron® and Primobolan® are resistant enough to breakdown to allow therapeutically beneficial blood levels to be achieved, although the overall bioavailability of these compounds is still much lower than methylated oral steroids.
  19. G_nome76

    Double barrel

    I draw my test and Tren in the same dart. Also planning on drawing TNE/Tren for pre workout. Can I mix the two ester types in the same dart or will that mess things up from a chemistry standpoint? Also are there other gear that should not be run in the same dart? Sent from my iPhone using Tapatalk
  20. Not sure if there is any science behind it but always pin in my shoulders over other areas. My thought is the higher on your body you pin the better dispersion you will get due to the help of gravity. Also it really blows up your shoulders arms and chest from what I've seen. Basically looking for where do you pin and why.
  21. glock4319

    Pre-workout AAS?

    What's up bros? Is there anything I can take to give me that monster feeling in the gym? I'm on a sust/winny cycle now so anything else that I could add to this for that extra lift? Sent from my iPhone using Tapatalk
  22. Pooty

    NPP and Deca

    So i got some NPP and some Deca comming. Question is am i right to think start the deca and maybe just the first 4 weeks use the npp till the deca gets seated in my system?? Dont worry im on test and winny, have AI and know all the sides. I just never ran npp before.
  23. So I'm going to layout my upcoming cut cycle, and as I currently have it, I. Plan on injecting monday/Wednesday/Friday. Ive ran 2 cycles with tren ace. Both at 300 mg per week. And I've always injected eod. Ive heard all the reasons to inject daily. Stable hormone levels in blood. Etc. Some of you guys/gals who have tried both, what's your thoughts? Mon- 1ml-tri tren200 1ml-test P 100 1ml- mast e200 Wed- 1ml-tren ace100 1ml-Test P100 Fri- 1ml-tren ace100 1ml-Test P100 1ml-Mast e200. Aromasin on hand if needed. And possibly some Dianabol thrown in if I get too flat. Possibly tossed in at 20-25mg daily. Also have like 30 ml of test suspension in oil. Possibly throw that in ?? Thoughts opinions? Should I be splitting these doses to inject daily? Should I add suspension? Open to any and all input. Thanks.
  24. Guest

    Body Builder Dies

    http://www.dailymail.co.uk/news/article-4319870/Bodybuilder-died-taking-steroids-dark-web.html
  25. READ THE RULES BEFORE POSTING: 1. Only domestic members are able to participate in the contest. 2. Contest starts Monday 3-27 and ends Friday 3-31 3. Must be Worst Cycle Experience 4. Discussion outside of the Worst Cycle Experience are NOT permitted. This will disqualify the member(s) from eligibility for the contest. All post outside the Worst Cycle Experience will be deleted. 5. Please keep your posts respectful(ish). 6. Each member is limited to 2 posts within 24 hours. Posts CANNOT be back to back, another member must comment before you can comment again. 7. Have fun - laugh a bit while we find a winner. This weeks sponsor is IAsuperpharma The winner of this weeks contest will receive $200 store credit towards the purchase of $200 or more IAsuperpharma gear, compliments of Anabolic Architects. My worst cycle experience was?
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